The Department of Physiology, Biochemistry and Pharmacology at the College of Veterinary Medicine has witnessed the discussion of the master’s thesis entitled (The effect of sodium butyrate supplement on the results of lead acetate induced for liver toxicity in rats) submitted by the student Rusul Muwafaq Ahmed. The aim of this study was that sodium butyrate, one of the metabolites, can reduce the toxic effect of lead acetate on the function of the liver, which is the main organ in the excretion of toxins out of the body. The study concluded that sodium butyrate has the potential to improve the harmful effect of lead acetate on liver function by significantly reducing inflammatory processes by increasing anti-inflammatory gene expression of Interleukin 10 (IL-10) and an increase of the antioxidant production associated with reduced malondidehyde production.
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